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Eyes Wide Open: A new model for tackling poverty and inequality
How might we begin tackling poverty and extreme inequality? There’s a simple answer: in the places that we know are doing best, and that makes the most sense. This has two benefits. Firstly, it means we can follow the programme that works. Secondly, it means that we don’t have to reinvent the wheel (because we already know it). So how about this: we should look at countries like the Nordic countries that are at the top end of inequality and poverty, and look at the good things they’ve done.
This is the approach that the REACH Strategy (Rebuilding Europe’s Attractiveness by Converting Globalisation into Growth) took. It looked at some of the areas that were strongest in the Nordic countries, and created a framework that could be applied not only in the Nordic countries, but in other regions where those things are working. But what is it?
REACH Strategy
REACH stands for ‘Rebuilding Europe’s Attractiveness by Converting Globalisation into Growth.’ It’s essentially about moving from an economy that is mainly based on trade and services to one that is based on producing value added products. Specifically, it means shifting from ‘aiding trade’ to ‘promoting economic growth and employment through trade’ – in other words, turning the existing multinational companies that the Nordic countries have (and which in most cases are owned by both domestic and global companies) into the companies that are producing the products that we want.
A third benefit of this approach to inequality and poverty reduction is that this means we don’t have to reinvent the wheel, because we already know what works. So in a nutshell: we should look at what the Nordic countries and other leading economies have done that works to tackle poverty and inequality, we should try to make this a model for other regions, and we should ensure that if it works elsewhere it can be scaled up. and in vivo data (including histological findings and decreased viral load) also support its use in combination with DAAs. Favipiravir has a broad-spectrum antiviral activity, but the low genetic barrier to resistance and potential for toxicities make it an unsuitable first-line therapy in practice. Trials have shown its promising results for the treatment of COVID-19, which is likely related to its multiple mechanisms of action \[[
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