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Previous studies suggested that high levels of plasma-free arachidonic acid (PUFA-AA) are associated with endothelial dysfunction and/or atherogenesis. The objectives of this study were 1) to determine whether plasma-free AA levels are increased in hypercholesterolemia and 2) to determine whether cultured endothelial cells (EC) from normal or hypercholesterolemic C57BL/6 mice can modulate these levels. AA content was determined in EC using gas chromatography, after incubation with [1-14C]AA. Plasma-free AA was increased in mice fed a cholesterol-rich diet (P 0.10) at baseline or after 6 mo of a cholesterol-rich diet. EC from hypercholesterolemic mice were able to restore their AA content to normal levels after incubation with AA compared with EC